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Query author contributions in Reactome

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Details on Person Stephan, Ralf, 2025-06-20

Class:IdInstanceEdit:9958441
_displayNameStephan, Ralf, 2025-06-20
_timestamp2025-06-20 08:00:19
author[Person:804962] Stephan, Ralf
dateTime2025-06-20 07:59:40
(created)[DatabaseIdentifier:9958439] RHEA:29352
[DatabaseIdentifier:9958440] RHEA:29353
[DatabaseIdentifier:9958442] RHEA:62253
(modified)[Reaction:437136] SLC30A8 transports Zn2+ from cytosol to secretory granule [Homo sapiens]
[Summation:437143] The human SLC30A8 gene encodes the zinc transporter ZnT8 whi...
[Reaction:442317] ZIP6 and ZIP14 mediate zinc influx into cells [Homo sapiens]
[Reaction:442345] hZIP10 mediates zinc influx into cells [Homo sapiens]
[Summation:442353] The human gene SLC39A8 encodes the zinc transporter ZIP8 (BI...
[Reaction:442387] ZIP8 mediates zinc influx into cells [Homo sapiens]
[Reaction:442393] ZIP7 mediates zinc efflux from the endoplasmic reticulum [Homo sapiens]
[Reaction:442405] hZIP5 mediates zinc uptake by cells [Homo sapiens]
[Reaction:442422] SLC39A1-4 transports Zn2+ from extracellular region to cytosol [Homo sapiens]
[PathwayDiagram:9957242] Diagram of Metal ion SLC transporters, Defective SLC9A6 causes X-linked, syndromic mental retardation,, Christianson type (MRXSCH), Defective SLC9A9 causes autism 16 (AUTS16), Defective SLC24A4 causes hypomineralized amelogenesis imperfecta (AI), Defective SLC24A5 causes oculocutaneous albinism 6 (OCA6), and Defective SLC39A4 causes acrodermatitis enteropathica, zinc-deficiency type (AEZ)
(structureModified)[Reaction:437136] SLC30A8 transports Zn2+ from cytosol to secretory granule [Homo sapiens]
[Reaction:442387] ZIP8 mediates zinc influx into cells [Homo sapiens]
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No pathways have been reviewed or authored by Stephan, Ralf, 2025-06-20 (9958441)
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