Reactome: A Curated Pathway Database
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Query author contributions in Reactome

Reactome depends on collaboration between our curation team and outside experts to assemble and peer-review its pathway modules. The integration of ORCID within Reactome enables us to meet a key challenge with authoring, curating and reviewing biological information by incentivizing and crediting the external experts that contribute their expertise and time to the Reactome curation process. More information is available at ORCID and Reactome.

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Details on Person Layton, J E

Class:IdPerson:9674569
_displayNameLayton, J E
_timestamp2020-01-15 16:34:27
created[InstanceEdit:9674559] May, Bruce, 2020-01-15
firstnameJ E
initialJE
surnameLayton
(author)[LiteratureReference:9674549] Tyrosine kinase JAK1 is associated with the granulocyte-colony-stimulating factor receptor and both become tyrosine-phosphorylated after receptor activation
[LiteratureReference:9674904] Tyrosine residues in the granulocyte colony-stimulating factor (G-CSF) receptor mediate G-CSF-induced differentiation of murine myeloid leukemic (M1) cells
[LiteratureReference:9676040] Distinct regions of the granulocyte colony-stimulating factor receptor are required for tyrosine phosphorylation of the signaling molecules JAK2, Stat3, and p42, p44MAPK
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No pathways have been reviewed or authored by Layton, J E (9674569)