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Details on Person Jassal, Bijay, 2019-09-18

Class:Id	InstanceEdit:9661797
_displayName	Jassal, Bijay, 2019-09-18
_timestamp	2019-09-18 14:22:39
author	[Person:73447] Jassal, Bijay
dateTime	2019-09-18 14:22:14
(created)	[Summation:9661796] Bilirubin (BIL), the end product of heme catabolism, is take... [CatalystActivity:9661798] secondary active transmembrane transporter activity of SLCO1B3 [plasma membrane] [Reaction:9661799] SLCO1B3 transports BIL from extracellular region (blood) to cytosol (hepatocyte) [Homo sapiens] [StableIdentifier:9661800] R-HSA-9661799.3
(modified)	[BlackBoxEvent:189381] BIL translocates from the cytosol to the ER lumen [Homo sapiens] [Pathway:189483] Heme degradation [Homo sapiens] [PathwayDiagram:421392] Diagram of Metabolism of porphyrins, Defective SLCO1B1 causes hyperbilirubinemia, Rotor type (HBLRR), Defective SLCO1B3 causes hyperbilirubinemia, Rotor type (HBLRR), Defective ABCC2 causes DJS, Defective UGT1A1 causes hyperbilirubinemia, and Defective UGT1A4 causes hyperbilirubinemia [Pathway:5619058] Defective SLCO1B3 causes hyperbilirubinemia, Rotor type (HBLRR) [Homo sapiens] [Pathway:5619110] Defective SLCO1B1 causes hyperbilirubinemia, Rotor type (HBLRR) [Homo sapiens] [Summation:5661179] In the body, solute carrier organic anion transporter family... [FailedReaction:5661184] Defective SLCO1B1 does not transport BIL from extracellular region (blood) to cytosol (hepatocyte) [Homo sapiens] [Summation:5661186] In the body, solute carrier organic anion transporter family... [Summation:5661187] The solute carrier organic anion transporter family member 1... [FailedReaction:5661198] Defective SLCO1B3 does not transport BIL from extracellular region (blood) to cytosol (hepatocyte) [Homo sapiens] List all 11 refering instances
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No pathways have been reviewed or authored by Jassal, Bijay, 2019-09-18 (9661797)