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Details on Person Jassal, Bijay, 2019-02-25

Class:IdInstanceEdit:9638118
_displayNameJassal, Bijay, 2019-02-25
_timestamp2019-02-25 17:25:54
author[Person:73447] Jassal, Bijay
dateTime2019-02-25 22:24:27
(created)[CatalystActivity:9638115] exo-alpha-sialidase activity of NEU4:GLB1:CTSA dimer [lysosomal lumen]
[Summation:9638116] Sialidases 1-4 (NEU1-4, neuraminidases, receptor-destroying ...
[Complex:9638117] NEU1 mutants:GLB1:CTSA dimer [lysosomal lumen] [Homo sapiens]
[CatalystActivity:9638119] exo-alpha-sialidase activity of NEU1 mutants:GLB1:CTSA dimer [lysosomal lumen]
[Reaction:9638120] NEU4 hydrolyses Neu5Ac from glycoconjugates [Homo sapiens]
[StableIdentifier:9638121] R-HSA-9638120.3
[StableIdentifier:9638122] R-HSA-9638117.1
(modified)[Complex:1605684] NEU1:GLB1:CTSA dimer [lysosomal lumen] [Homo sapiens]
[Reaction:4084999] NEU1 hydrolyses Neu5Ac from glycoconjugates [Homo sapiens]
[Pathway:4085001] Sialic acid metabolism [Homo sapiens]
[CatalystActivity:4088012] exo-alpha-sialidase activity of NEU1:GLB1:CTSA dimer [lysosomal lumen]
[Complex:4088186] NEU4:GLB1:CTSA dimer [lysosomal lumen] [Homo sapiens]
[FailedReaction:4341669] Defective NEU1 does not hydrolyse Neu5Ac from glycoconjugates [Homo sapiens]
[PathwayDiagram:4594867] Diagram of Sialic acid metabolism, Defective SLC17A5 causes Salla disease (SD) and ISSD, Defective NEU1 causes sialidosis, Defective GNE causes sialuria, NK and IBM2, and Defective SLC35A1 causes congenital disorder of glycosylation 2F (CDG2F)
[EntityFunctionalStatus:5674192] loss_of_function of NEU1 mutants:GLB1:CTSA dimer [lysosomal lumen]
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No pathways have been reviewed or authored by Jassal, Bijay, 2019-02-25 (9638118)
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