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Query author contributions in Reactome

Reactome depends on collaboration between our curation team and outside experts to assemble and peer-review its pathway modules. The integration of ORCID within Reactome enables us to meet a key challenge with authoring, curating and reviewing biological information by incentivizing and crediting the external experts that contribute their expertise and time to the Reactome curation process. More information is available at ORCID and Reactome.

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Details on Person Orlic-Milacic, Marija, 2018-12-24

Class:IdInstanceEdit:9632937
_displayNameOrlic-Milacic, Marija, 2018-12-24
_timestamp2018-12-24 16:42:42
author[Person:1169272] Orlic-Milacic, Marija
dateTime2018-12-24 21:42:33
(authored)[Pathway:9630747] Diseases of Cellular Senescence [Homo sapiens]
[Pathway:9630750] Evasion of Oncogene Induced Senescence Due to p16INK4A Defects [Homo sapiens]
[Pathway:9630791] Evasion of Oncogene Induced Senescence Due to Defective p16INK4A binding to CDK4 [Homo sapiens]
[FailedReaction:9630792] p16INK4A mutants do not bind CDK4 [Homo sapiens]
[Pathway:9630794] Evasion of Oncogene Induced Senescence Due to Defective p16INK4A binding to CDK4 and CDK6 [Homo sapiens]
[FailedReaction:9630795] p16INK4A mutants do not bind CDK4,CDK6 [Homo sapiens]
[Pathway:9632693] Evasion of Oxidative Stress Induced Senescence Due to p16INK4A Defects [Homo sapiens]
[Pathway:9632697] Evasion of Oxidative Stress Induced Senescence Due to Defective p16INK4A binding to CDK4 [Homo sapiens]
[Pathway:9632700] Evasion of Oxidative Stress Induced Senescence Due to Defective p16INK4A binding to CDK4 and CDK6 [Homo sapiens]
(modified)[Pathway:9630750] Evasion of Oncogene Induced Senescence Due to p16INK4A Defects [Homo sapiens]
[Summation:9630755] The CDKN2A gene consists of four exons, exon 1beta, exon 1al...
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No pathways have been reviewed or authored by Orlic-Milacic, Marija, 2018-12-24 (9632937)