Reactome: A Curated Pathway Database
THIS SITE IS USED FOR CURATION AND TESTING
IT IS NOT STABLE, IS LINKED TO AN INCOMPLETE DATA SET, AND IS NOT MONITORED FOR PERFORMANCE. WE STRONGLY RECOMMEND THE USE OF OUR PUBLIC SITE

Query author contributions in Reactome

Reactome depends on collaboration between our curation team and outside experts to assemble and peer-review its pathway modules. The integration of ORCID within Reactome enables us to meet a key challenge with authoring, curating and reviewing biological information by incentivizing and crediting the external experts that contribute their expertise and time to the Reactome curation process. More information is available at ORCID and Reactome.

If you have an ORCID ID that is not listed on this page, please forward this information to us and we will update your Reactome pathway records.

Name Email address

Details on Person CDKN2A/p16 is inactivated in most melanoma cell lines

Class:IdLiteratureReference:9632578
_displayNameCDKN2A/p16 is inactivated in most melanoma cell lines
_timestamp2018-12-19 21:11:52
author[Person:9631249] Castellano, M
[Person:9632577] Pollock, P M
[Person:9632576] Walters, M K
[Person:9632575] Sparrow, L E
[Person:9632579] Down, L M
[Person:9631246] Gabrielli, B G
[Person:9632580] Parsons, P G
[Person:9631250] Hayward, Nicholas K
created[InstanceEdit:9632581] Orlic-Milacic, Marija, 2018-12-19
journalCancer Res.
pages4868-75
pubMedIdentifier9354451
titleCDKN2A/p16 is inactivated in most melanoma cell lines
volume57
year1997
(literatureReference)[EntityWithAccessionedSequence:9632582] p16INK4A R58* [cytosol] [Homo sapiens]
[EntityWithAccessionedSequence:9632585] p16INK4A W110* [cytosol] [Homo sapiens]
[EntityWithAccessionedSequence:9632586] p16INK4A R80* [cytosol] [Homo sapiens]
[EntityWithAccessionedSequence:9632587] p16INK4A E120* [cytosol] [Homo sapiens]
[EntityWithAccessionedSequence:9632588] p16INK4A E88* [cytosol] [Homo sapiens]
[Pathway:9630750] Evasion of Oncogene Induced Senescence Due to p16INK4A Defects [Homo sapiens]
[FailedReaction:9630795] p16INK4A mutants do not bind CDK4,CDK6 [Homo sapiens]
[Pathway:9632693] Evasion of Oxidative Stress Induced Senescence Due to p16INK4A Defects [Homo sapiens]
[Change default viewing format]
No pathways have been reviewed or authored by CDKN2A/p16 is inactivated in most melanoma cell lines (9632578)
Follow Reactome