Reactome: A Curated Pathway Database
THIS SITE IS USED FOR CURATION AND TESTING
IT IS NOT STABLE, IS LINKED TO AN INCOMPLETE DATA SET, AND IS NOT MONITORED FOR PERFORMANCE. WE STRONGLY RECOMMEND THE USE OF OUR PUBLIC SITE

Query author contributions in Reactome

Reactome depends on collaboration between our curation team and outside experts to assemble and peer-review its pathway modules. The integration of ORCID within Reactome enables us to meet a key challenge with authoring, curating and reviewing biological information by incentivizing and crediting the external experts that contribute their expertise and time to the Reactome curation process. More information is available at ORCID and Reactome.

If you have an ORCID ID that is not listed on this page, please forward this information to us and we will update your Reactome pathway records.

Name Email address

Details on Person Human PEX1 cloned by functional complementation on a CHO cell mutant is responsible for peroxisome-deficient Zellweger syndrome of complementation group I

Class:IdLiteratureReference:9033849
_displayNameHuman PEX1 cloned by functional complementation on a CHO cell mutant is responsible for peroxisome-deficient Zellweger syndrome of complementation group I
_timestamp2017-12-28 17:57:22
author[Person:9033835] Tamura, S
[Person:9033872] Okumoto, K
[Person:9033842] Toyama, R
[Person:2458314] Shimozawa, N
[Person:77531] Tsukamoto, T
[Person:2285500] Suzuki, Y
[Person:5320202] Osumi, T
[Person:2458321] Kondo, N
[Person:5321239] Fujiki, Y
created[InstanceEdit:9033817] May, Bruce, 2017-12-28
journalProc. Natl. Acad. Sci. U.S.A.
pages4350-5
pubMedIdentifier9539740
titleHuman PEX1 cloned by functional complementation on a CHO cell mutant is responsible for peroxisome-deficient Zellweger syndrome of complementation group I
volume95
year1998
(literatureReference)[BlackBoxEvent:9033499] PEX1:PEX6:PEX26:ZFAND6 dissociates Ub:PEX5L and PEX7 from PEX14:PEX13:PEX2:PEX10:PEX12 and translocates PEX5L and PEX7 from the peroxisomal membrane to the cytosol [Homo sapiens]
[BlackBoxEvent:9033505] PEX1:PEX6:PEX26:ZFAND6:Ub:PEX5S,L:PEX14:PEX13:PEX2:PEX10:PEX12 dissociates yielding cytosolic Ub:PEX5S,L and membrane PEX14:PEX13:PEX2:PEX10:PEX12 [Homo sapiens]
[Change default viewing format]
No pathways have been reviewed or authored by Human PEX1 cloned by functional complementation on a CHO cell mutant is responsible for peroxisome-deficient Zellweger syndrome of complementation group I (9033849)