Reactome: A Curated Pathway Database
THIS SITE IS USED FOR CURATION AND TESTING
IT IS NOT STABLE, IS LINKED TO AN INCOMPLETE DATA SET, AND IS NOT MONITORED FOR PERFORMANCE. WE STRONGLY RECOMMEND THE USE OF OUR PUBLIC SITE

Query author contributions in Reactome

Reactome depends on collaboration between our curation team and outside experts to assemble and peer-review its pathway modules. The integration of ORCID within Reactome enables us to meet a key challenge with authoring, curating and reviewing biological information by incentivizing and crediting the external experts that contribute their expertise and time to the Reactome curation process. More information is available at ORCID and Reactome.

If you have an ORCID ID that is not listed on this page, please forward this information to us and we will update your Reactome pathway records.

Name Email address

Details on Person Sustained E2F-Dependent Transcription Is a Key Mechanism to Prevent Replication-Stress-Induced DNA Damage

Class:IdLiteratureReference:9007093
_displayNameSustained E2F-Dependent Transcription Is a Key Mechanism to Prevent Replication-Stress-Induced DNA Damage
_timestamp2017-05-26 14:40:11
author[Person:9007095] Bertoli, Cosetta
[Person:9007090] Herlihy, Anna E
[Person:9007094] Pennycook, Betheney R
[Person:6811107] Kriston-Vizi, Janos
[Person:9007096] de Bruin, Robertus A M
created[InstanceEdit:9007097] Orlic-Milacic, Marija, 2017-05-26
journalCell Rep
pages1412-22
pubMedIdentifier27160911
titleSustained E2F-Dependent Transcription Is a Key Mechanism to Prevent Replication-Stress-Induced DNA Damage
volume15
year2016
(literatureReference)[Summation:9007535] CHEK1 (Chk1), activated by ATR in response to replication st...
[Pathway:8953750] Transcriptional Regulation by E2F6 [Homo sapiens]
[Change default viewing format]
No pathways have been reviewed or authored by Sustained E2F-Dependent Transcription Is a Key Mechanism to Prevent Replication-Stress-Induced DNA Damage (9007093)