Reactome: A Curated Pathway Database
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Query author contributions in Reactome

Reactome depends on collaboration between our curation team and outside experts to assemble and peer-review its pathway modules. The integration of ORCID within Reactome enables us to meet a key challenge with authoring, curating and reviewing biological information by incentivizing and crediting the external experts that contribute their expertise and time to the Reactome curation process. More information is available at ORCID and Reactome.

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Details on Person Bonner, WM

Class:IdPerson:75768
_displayNameBonner, WM
_timestamp2003-09-01 15:51:33
firstnameWilliam M
initialWM
surnameBonner
(author)[LiteratureReference:75188] Histone H2AX phosphorylation is dispensable for the initial recognition of DNA breaks.
[LiteratureReference:83547] A critical role for histone H2AX in recruitment of repair factors to nuclear foci after DNA damage.
[LiteratureReference:83590] DNA double-stranded breaks induce histone H2AX phosphorylation on serine 139.
[LiteratureReference:217143] Collaborative roles of gammaH2AX and the Rad51 paralog Xrcc3 in homologous recombinational repair
[LiteratureReference:913249] Recombinational DNA double-strand breaks in mice precede synapsis
[LiteratureReference:3785751] Distribution and dynamics of chromatin modification induced by a defined DNA double-strand break
[LiteratureReference:5682460] Genomic instability in mice lacking histone H2AX
[LiteratureReference:5683400] DNA damage-induced G2-M checkpoint activation by histone H2AX and 53BP1
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No pathways have been reviewed or authored by Bonner, WM (75768)