Query author contributions in Reactome

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Pathways reviewed by Baer, Richard J (5659786)

DB_ID	Name
9699150	Defective DNA double strand break response due to BARD1 loss of function
9663199	Defective DNA double strand break response due to BRCA1 loss of function

Details on Person Baer, Richard J

Class:Id	Person:5659786
_displayName	Baer, Richard J
_timestamp	2020-11-16 22:06:44
affiliation	[Affiliation:9707401] Columbia University, Department of Pathology and Cell Biology
created	[InstanceEdit:5659776] Orlic-Milacic, Marija, 2014-12-23
firstname	Richard J
initial	RJ
modified	[InstanceEdit:9706945] Orlic-Milacic, Marija, 2020-11-09 [InstanceEdit:9707402] Orlic-Milacic, Marija, 2020-11-16
surname	Baer
(author)	[LiteratureReference:5659777] Structural requirements for the BARD1 tumor suppressor in chromosomal stability and homology-directed DNA repair [LiteratureReference:5659798] Loss of Bard1, the heterodimeric partner of the Brca1 tumor suppressor, results in early embryonic lethality and chromosomal instability [LiteratureReference:5659799] BARD1 participates with BRCA1 in homology-directed repair of chromosome breaks [LiteratureReference:5659807] The basal-like mammary carcinomas induced by Brca1 or Bard1 inactivation implicate the BRCA1/BARD1 heterodimer in tumor suppression [LiteratureReference:5662732] Tumor suppression in the absence of p53-mediated cell-cycle arrest, apoptosis, and senescence [LiteratureReference:5684129] Damage-induced BRCA1 phosphorylation by Chk2 contributes to the timing of end resection [LiteratureReference:8856576] Ferroptosis as a p53-mediated activity during tumour suppression [LiteratureReference:9700982] E3 ligase activity of BRCA1 is not essential for mammalian cell viability or homology-directed repair of double-strand DNA breaks [LiteratureReference:9700997] The BRCA1/BARD1 heterodimer assembles polyubiquitin chains through an unconventional linkage involving lysine residue K6 of ubiquitin [InstanceEdit:9706944] Baer, Richard J, 2020-11-04
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