Reactome: A Curated Pathway Database
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Query author contributions in Reactome

Reactome depends on collaboration between our curation team and outside experts to assemble and peer-review its pathway modules. The integration of ORCID within Reactome enables us to meet a key challenge with authoring, curating and reviewing biological information by incentivizing and crediting the external experts that contribute their expertise and time to the Reactome curation process. More information is available at ORCID and Reactome.

If you have an ORCID ID that is not listed on this page, please forward this information to us and we will update your Reactome pathway records.

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Details on Person goiter

Class:IdDisease:5627854
_displayNamegoiter
_timestamp2014-10-23 08:15:57
created[InstanceEdit:5627846] Jassal, Bijay, 2014-10-23
definitionA thyroid gland disease that involves an abnormal enlargement of the thyroid gland.
identifier12176
namegoiter
referenceDatabase[ReferenceDatabase:1247631] DOID
(disease)[EntityWithAccessionedSequence:5627850] SLC26A4 T416P [plasma membrane] [Homo sapiens]
[EntityWithAccessionedSequence:5627857] SLC26A4 L236P [plasma membrane] [Homo sapiens]
[EntityWithAccessionedSequence:5627860] SLC26A4 E384G [plasma membrane] [Homo sapiens]
[EntityWithAccessionedSequence:5627865] SLC26A4 H723R [plasma membrane] [Homo sapiens]
[DefinedSet:5627871] SLC26A4 mutants [plasma membrane] [Homo sapiens]
[EntityWithAccessionedSequence:5627873] SLC26A4 F667C [plasma membrane] [Homo sapiens]
[Pathway:5619046] Defective SLC26A4 causes Pendred syndrome (PDS) [Homo sapiens]
[FailedReaction:5627870] SLC26A4 does not transport I- from cytosol to extracellular region [Homo sapiens]
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No pathways have been reviewed or authored by goiter (5627854)