Reactome: A Curated Pathway Database
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Query author contributions in Reactome

Reactome depends on collaboration between our curation team and outside experts to assemble and peer-review its pathway modules. The integration of ORCID within Reactome enables us to meet a key challenge with authoring, curating and reviewing biological information by incentivizing and crediting the external experts that contribute their expertise and time to the Reactome curation process. More information is available at ORCID and Reactome.

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Details on Person MYD88 recruits unphosphorylated, inactive IRAK1 to the IL1 r...

Class:IdSummation:450116
_displayNameMYD88 recruits unphosphorylated, inactive IRAK1 to the IL1 r...
_timestamp2009-12-14 17:39:17
created[InstanceEdit:450128] Jupe, S, 2009-12-14
textMYD88 recruits unphosphorylated, inactive IRAK1 to the IL1 receptor complex.
(summation)[Reaction:446692] IRAK1 binds to MYD88 within the IL1R complex [Homo sapiens]
[Reaction:975115] IRAK1/or IRAK2 binds to the activated IRAK4 :MyD88:activated TLR 7/8 or 9 [Homo sapiens]
[Reaction:975852] IRAK1/or IRAK2 binds to the activated IRAK4 :oligo MyD88:activated TLR5 or 10 complex [Homo sapiens]
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No pathways have been reviewed or authored by MYD88 recruits unphosphorylated, inactive IRAK1 to the IL1 r... (450116)