Reactome: A Curated Pathway Database
THIS SITE IS USED FOR CURATION AND TESTING
IT IS NOT STABLE, IS LINKED TO AN INCOMPLETE DATA SET, AND IS NOT MONITORED FOR PERFORMANCE. WE STRONGLY RECOMMEND THE USE OF OUR PUBLIC SITE

Query author contributions in Reactome

Reactome depends on collaboration between our curation team and outside experts to assemble and peer-review its pathway modules. The integration of ORCID within Reactome enables us to meet a key challenge with authoring, curating and reviewing biological information by incentivizing and crediting the external experts that contribute their expertise and time to the Reactome curation process. More information is available at ORCID and Reactome.

If you have an ORCID ID that is not listed on this page, please forward this information to us and we will update your Reactome pathway records.

Name Email address

Details on Person Clinical characteristics and biochemical mechanisms of congenital hyperinsulinism associated with dominant KATP channel mutations

Class:IdLiteratureReference:445969
_displayNameClinical characteristics and biochemical mechanisms of congenital hyperinsulinism associated with dominant KATP channel mutations
_timestamp2009-11-07 07:27:09
author[Person:445942] Pinney, SE
[Person:445975] MacMullen, C
[Person:207476] Becker, S
[Person:445972] Lin, YW
[Person:445962] Hanna, C
[Person:445943] Thornton, P
[Person:445963] Ganguly, A
[Person:445978] Shyng, SL
[Person:77297] Stanley, CA
created[InstanceEdit:445970] May, B, 2009-11-07
journalJ Clin Invest
modified[InstanceEdit:445988] May, B, 2009-11-07
pages2877-86
pubMedIdentifier18596924
titleClinical characteristics and biochemical mechanisms of congenital hyperinsulinism associated with dominant KATP channel mutations
volume118
year2008
(literatureReference)[Reaction:265682] KCNJ11 tetramer:ABCC8 tetramer binds 4xATP, closing the channel [Homo sapiens]
[Change default viewing format]
No pathways have been reviewed or authored by Clinical characteristics and biochemical mechanisms of congenital hyperinsulinism associated with dominant KATP channel mutations (445969)