Query author contributions in Reactome
Reactome depends on collaboration between our curation team and outside experts to assemble and peer-review its pathway modules. The integration of ORCID within Reactome enables us to meet a key challenge with authoring, curating and reviewing biological information by incentivizing and crediting the external experts that contribute their expertise and time to the Reactome curation process. More information is available at ORCID and Reactome.
If you have an ORCID ID that is not listed on this page, please forward this information to us and we will update your Reactome pathway records.
Details on Person CCR9 (previously called orphan receptor GPR 9-6) is highly e...
| Class:Id | Summation:373250 |
|---|---|
| _displayName | CCR9 (previously called orphan receptor GPR 9-6) is highly e... |
| _timestamp | 2008-07-15 13:28:46 |
| created | [InstanceEdit:373311] Jassal, Bijay, 2008-07-15 |
| modified | [InstanceEdit:373332] Jassal, B, 2008-07-15 13:27:23 |
| text | CCR9 (previously called orphan receptor GPR 9-6) is highly expressed in the thymus on both mature and immature T-cells. It is also abundant in T-cells of the intestine but is lowly expressed in lymph nodes and the spleen. Alternative splicing produces two receptors, called CCR9A and CCR9B, CCR9A containing 12 additional amino acids at its N terminus as compared with CCR9B (Yu CR et al, 2000). The A and B forms of the receptor were expressed at a ratio of approximately 10:1. CCR9 has a specific ligand in CCL25 (Thymus-expressed chemokine, TECK) (Zaballos A et al, 1999) and transduces the signal by intracellular calcium mobilization. |
| (summation) | [Reaction:373074] Receptor CCR9 binds CCL25 ligand [Homo sapiens] |
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No pathways have been reviewed or authored by CCR9 (previously called orphan receptor GPR 9-6) is highly e... (373250)
