Query author contributions in Reactome
Reactome depends on collaboration between our curation team and outside experts to assemble and peer-review its pathway modules. The integration of ORCID within Reactome enables us to meet a key challenge with authoring, curating and reviewing biological information by incentivizing and crediting the external experts that contribute their expertise and time to the Reactome curation process. More information is available at ORCID and Reactome.
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Details on Person E2F1, E2F2 or E2F3 in complex with SP1 stimulates p14ARF tra...
| Class:Id | Summation:3245957 |
|---|---|
| _displayName | E2F1, E2F2 or E2F3 in complex with SP1 stimulates p14ARF tra... |
| _timestamp | 2019-05-06 20:30:46 |
| created | [InstanceEdit:3245954] Orlic-Milacic, M, 2013-03-28 |
| modified | [InstanceEdit:3299672] Orlic-Milacic, M, 2013-04-20 [InstanceEdit:8979102] Orlic-Milacic, Marija, 2017-02-21 [InstanceEdit:9645195] Orlic-Milacic, Marija, 2019-05-06 |
| text | E2F1, E2F2 or E2F3 in complex with SP1 stimulates p14ARF transcription (Parisi et al. 2002). Therefore, increased activity of E2F1, E2F2 or E2F3, which may result from the loss of function of the tumor suppressor protein RB1, an inhibitor of E2F1/2/3, leads to an increased level of p14ARF (Komori et al. 2005). |
| (summation) | [BlackBoxEvent:3209109] E2F1/E2F2/E2F3 and SP1 stimulate transcription of p14ARF mRNA [Homo sapiens] |
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No pathways have been reviewed or authored by E2F1, E2F2 or E2F3 in complex with SP1 stimulates p14ARF tra... (3245957)
