Reactome: A Curated Pathway Database
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Query author contributions in Reactome

Reactome depends on collaboration between our curation team and outside experts to assemble and peer-review its pathway modules. The integration of ORCID within Reactome enables us to meet a key challenge with authoring, curating and reviewing biological information by incentivizing and crediting the external experts that contribute their expertise and time to the Reactome curation process. More information is available at ORCID and Reactome.

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Details on Person Newton, Kim

Class:IdPerson:2586698
_displayNameNewton, Kim
_timestamp2012-11-16 09:48:03
created[InstanceEdit:2586708] Shamovsky, V, 2012-11-16
firstnameKim
initialK
surnameNewton
(author)[LiteratureReference:2586701] Ubiquitin chain editing revealed by polyubiquitin linkage-specific antibodies
[LiteratureReference:5621050] Activity of protein kinase RIPK3 determines whether cells die by necroptosis or apoptosis
[LiteratureReference:5690747] Loss of the tumor suppressor BAP1 causes myeloid transformation
[LiteratureReference:9652835] Non-canonical inflammasome activation targets caspase-11
[LiteratureReference:9687059] Cleavage of RIPK1 by caspase-8 is crucial for limiting apoptosis and necroptosis
[LiteratureReference:9698306] Kinase domain dimerization drives RIPK3-dependent necroptosis
[LiteratureReference:9757942] Integration of innate immune signalling by caspase-8 cleavage of N4BP1
[LiteratureReference:9817349] Impaired RIPK1 ubiquitination sensitizes mice to TNF toxicity and inflammatory cell death
[LiteratureReference:9819072] Autophagy regulates inflammatory programmed cell death via turnover of RHIM-domain proteins
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No pathways have been reviewed or authored by Newton, Kim (2586698)