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Details on Person UniProt:Q9H792 PEAK1

Class:IdReferenceGeneProduct:248091
_chainChangeLogchain:1-1746 added on Fri February 6 2015
_displayNameUniProt:Q9H792 PEAK1
_timestamp2024-11-03 19:53:09
chainchain:1-1746
checksum7C9A9F60BBCF6C61
commentFUNCTION Probable catalytically inactive kinase. Scaffolding protein that regulates the cytoskeleton to control cell spreading and migration by modulating focal adhesion dynamics (PubMed:20534451, PubMed:23105102, PubMed:35687021). Acts as a scaffold for mediating EGFR signaling (PubMed:23846654).SUBUNIT Homodimer (PubMed:29212708). Interacts with BCAR1 and CRK (PubMed:20534451). Interacts with PRAG1 (PubMed:29079850). Interacts (when phosphorylated at Tyr-1188) with SHC1 (via PID domain) (PubMed:23846654, PubMed:35687021). Found in a complex with PPP1CA, PPP1CC, SHC1 and PEAK1. Interacts (when phosphorylated at Tyr-635) with tensin TNS3 (when phosphorylated on the SH2 domain); TNS3 also interacts with integrins ITGB1, ITGB3 and ITGB5 and mediates their association with PEAK1 (PubMed:35687021). Interacts with RASAL2 and GRB2 (PubMed:35687021).INTERACTION Colocalizes with F-actin in serum-rich medium (PubMed:20534451). Actin colocalization is reduced during serum starvation (PubMed:20534451).DOMAIN The dimerization region encompasses helices both from the N- and C-terminal of the protein kinase domain.PTM Phosphorylated on tyrosine in a CSK-dependent manner in response to adhesion to fibronectin and to EGF stimulation (PubMed:20534451). Phosphorylation at Tyr-665 by a Src family kinase controls subcellular localization to focal adhesion and focal adhesion dynamics (PubMed:20534451). Phosphorylation at Tyr-1188 is essential for binding to SHC1 (PubMed:23846654). Phosphorylation at Tyr-635 promotes interaction with tensin TNS3 (PubMed:35687021).SIMILARITY Belongs to the protein kinase superfamily.CAUTION Has been the subject of controversy surrounding its catalytic capabilities. Early characterization of PEAK1 gave a weak in vitro tyrosine kinase activity (PubMed:20534451). The crystal structure indicates that the kinase-domain contains a closed nucleotide-binding cleft that in this conformation may deleteriously affect nucleotide binding (PubMed:29212708). Furthermore PEAK1 is devoid of nucleotide binding activity, as detected by a thermal-shift assay (PubMed:24107129). So it seems probable that PEAK1 is an inactive kinase.
created[InstanceEdit:217385] Schmidt, EE, 2008-03-27 06:23:53
descriptionrecommendedName: fullName evidence="14"Inactive tyrosine-protein kinase PEAK1 alternativeName: fullName evidence="12"Pseudopodium-enriched atypical kinase 1 alternativeName: fullName evidence="13"Sugen kinase 269 alternativeName: Tyrosine-protein kinase SgK269
geneNamePEAK1
KIAA2002
identifierQ9H792
isSequenceChangedFALSE
keyword3D-structure
Cell junction
Cytoplasm
Cytoskeleton
Phosphoprotein
Proteomics identification
Reference proteome
modified[InstanceEdit:9836292] Weiser, Joel, 2023-05-25
[InstanceEdit:9852000] Weiser, Joel, 2023-11-03
[InstanceEdit:9917590] Weiser, Joel, 2024-08-09
[InstanceEdit:9926675] Weiser, Joel, 2024-11-03
namePEAK1
referenceDatabase[ReferenceDatabase:2] UniProt
referenceGene[ReferenceDNASequence:8988574] ENSEMBL:ENSG00000173517 PEAK1 [Homo sapiens]
secondaryIdentifierPEAK1_HUMAN
Q6ZS78
Q8NAZ4
Q8NCM3
Q8TEG7
sequenceLength1746
species[Species:48887] Homo sapiens
(referenceEntity)[EntityWithAccessionedSequence:9715162] PEAK1 [cytosol] [Homo sapiens]
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No pathways have been reviewed or authored by UniProt:Q9H792 PEAK1 (248091)