Query author contributions in Reactome

Reactome depends on collaboration between our curation team and outside experts to assemble and peer-review its pathway modules. The integration of ORCID within Reactome enables us to meet a key challenge with authoring, curating and reviewing biological information by incentivizing and crediting the external experts that contribute their expertise and time to the Reactome curation process. More information is available at ORCID and Reactome.

If you have an ORCID ID that is not listed on this page, please forward this information to us and we will update your Reactome pathway records.

Details on Person Boggon, TJ

Class:Id	Person:202244
_displayName	Boggon, TJ
_timestamp	2011-01-18 18:18:25
created	[InstanceEdit:202250] Garapati, P V, 2007-10-29 14:08:25
firstname	Titus J
initial	TJ
modified	[InstanceEdit:1169413] Orlic-Milacic, Marija, 2011-01-18
surname	Boggon
(author)	[LiteratureReference:202290] Structure and regulation of Src family kinases [LiteratureReference:1169414] Structures of lung cancer-derived EGFR mutants and inhibitor complexes: mechanism of activation and insights into differential inhibitor sensitivity [LiteratureReference:1228027] EGFR mutations in lung cancer: correlation with clinical response to gefitinib therapy [LiteratureReference:2534161] C-cadherin ectodomain structure and implications for cell adhesion mechanisms [LiteratureReference:5610177] G-protein signaling through tubby proteins [LiteratureReference:9652716] Exome sequencing identifies recurrent somatic RAC1 mutations in melanoma [LiteratureReference:9691838] Identification of driver and passenger mutations of FLT3 by high-throughput DNA sequence analysis and functional assessment of candidate alleles [LiteratureReference:9691847] Identifying and characterizing a novel activating mutation of the FLT3 tyrosine kinase in AML [LiteratureReference:9694111] PAK6 targets to cell-cell adhesions through its N-terminus in a Cdc42-dependent manner to drive epithelial colony escape [LiteratureReference:9854422] Lysine acetylation restricts mutant IDH2 activity to optimize transformation in AML cells
[Change default viewing format]

No pathways have been reviewed or authored by Boggon, TJ (202244)