Query author contributions in Reactome

Reactome depends on collaboration between our curation team and outside experts to assemble and peer-review its pathway modules. The integration of ORCID within Reactome enables us to meet a key challenge with authoring, curating and reviewing biological information by incentivizing and crediting the external experts that contribute their expertise and time to the Reactome curation process. More information is available at ORCID and Reactome.

If you have an ORCID ID that is not listed on this page, please forward this information to us and we will update your Reactome pathway records.

Details on Person TSC2 is phosphorylated and inhibited by Akt and suppresses mTOR signalling

Class:Id	LiteratureReference:199381
_displayName	TSC2 is phosphorylated and inhibited by Akt and suppresses mTOR signalling
_timestamp	2007-07-10 08:11:09
author	[Person:165677] Inoki, K [Person:68475] Li, Yun [Person:199378] Zhu, T [Person:180291] Wu, J [Person:111055] Guan, KL
created	[InstanceEdit:199370] Jassal, B, 2007-07-10 08:08:36
journal	Nat Cell Biol
pages	648-57
pubMedIdentifier	12172553
title	TSC2 is phosphorylated and inhibited by Akt and suppresses mTOR signalling
volume	4
year	2002
(literatureReference)	[Summation:166178] Tuberous sclerosis complex (TSC) is a multi-system genetic d... [Summation:179081] AKT (PKB) phosphorylates TSC2 decreasing its activity and di... [Summation:2399980] AKT1 E17K gain-of-function mutant is expected to phosphoryla... [Reaction:165162] Phosphorylation of TSC2 by AKT (PKB) [Homo sapiens] [Pathway:165181] Inhibition of TSC complex formation by AKT (PKB) [Homo sapiens] [Reaction:198609] AKT phosphorylates TSC2, inhibiting it [Homo sapiens] [Reaction:2399982] AKT1 E17K mutant phosphorylates TSC2, inhibiting it [Homo sapiens]
[Change default viewing format]

No pathways have been reviewed or authored by TSC2 is phosphorylated and inhibited by Akt and suppresses mTOR signalling (199381)