Query author contributions in Reactome
Reactome depends on collaboration between our curation team and outside experts to assemble and peer-review its pathway modules. The integration of ORCID within Reactome enables us to meet a key challenge with authoring, curating and reviewing biological information by incentivizing and crediting the external experts that contribute their expertise and time to the Reactome curation process. More information is available at ORCID and Reactome.
If you have an ORCID ID that is not listed on this page, please forward this information to us and we will update your Reactome pathway records.
Details on Person After activation, FGFR mutants are presumed to recruit FRS2 ...
| Class:Id | Summation:1982075 |
|---|---|
| _displayName | After activation, FGFR mutants are presumed to recruit FRS2 ... |
| _timestamp | 2014-12-03 20:42:26 |
| created | [InstanceEdit:1982076] Rothfels, K, 2011-11-09 |
| modified | [InstanceEdit:2085551] Rothfels, K, 2012-02-03 [InstanceEdit:2085637] Rothfels, K, 2012-02-05 [InstanceEdit:5654389] Rothfels, Karen, 2014-12-03 |
| text | After activation, FGFR mutants are presumed to recruit FRS2 (also known as FRS2alpha). This has been demonstrated in some cases (see for instance Ahmed, 2008; Weiss, 2010; Dutt, 2008; Dutt, 2011; Cha, 2009; Qing, 2009; Bai, 2010 ) and is inferred to occur in others by analogy with the wild-type receptor. |
| (summation) | [Reaction:5655269] Activated FGFR1 mutants bind FRS2 [Homo sapiens] [Reaction:5655339] Activated FGFR2 mutants bind FRS2 [Homo sapiens] [Reaction:5655351] Activated FGFR4 mutants bind FRS2 [Homo sapiens] |
| [Change default viewing format] | |
No pathways have been reviewed or authored by After activation, FGFR mutants are presumed to recruit FRS2 ... (1982075)
