Reactome: A Curated Pathway Database
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Query author contributions in Reactome

Reactome depends on collaboration between our curation team and outside experts to assemble and peer-review its pathway modules. The integration of ORCID within Reactome enables us to meet a key challenge with authoring, curating and reviewing biological information by incentivizing and crediting the external experts that contribute their expertise and time to the Reactome curation process. More information is available at ORCID and Reactome.

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Details on Person Dikic, I

Class:IdPerson:182983
_displayNameDikic, I
_timestamp2006-07-21 10:21:27
created[InstanceEdit:183010] Jassal, B, 2006-07-21 10:19:47
initialI
surnameDikic
(author)[LiteratureReference:182989] Cbl escapes Cdc42-mediated inhibition by downregulation of the adaptor molecule betaPix
[LiteratureReference:183032] Sprouty2 acts at the Cbl/CIN85 interface to inhibit epidermal growth factor receptor downregulation
[LiteratureReference:183056] Cbl-CIN85-endophilin complex mediates ligand-induced downregulation of EGF receptors
[LiteratureReference:183081] Tyrosine phosphorylation of the c-cbl proto-oncogene protein product and association with epidermal growth factor (EGF) receptor upon EGF stimulation
[LiteratureReference:188579] Cbl-directed monoubiquitination of CIN85 is involved in regulation of ligand-induced degradation of EGF receptors
[LiteratureReference:188597] Mechanisms controlling EGF receptor endocytosis and degradation
[LiteratureReference:190942] Identification of six novel autophosphorylation sites on fibroblast growth factor receptor 1 and elucidation of their importance in receptor activation and signal transduction
[LiteratureReference:430276] SH3P2 in complex with Cbl and Src
[LiteratureReference:433880] Involvement of the ubiquitin-like domain of TBK1/IKK-i kinases in regulation of IFN-inducible genes
[LiteratureReference:508442] Pyk2 is a downstream mediator of the IL-2 receptor-coupled Jak signaling pathway
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No pathways have been reviewed or authored by Dikic, I (182983)