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Query author contributions in Reactome

Reactome depends on collaboration between our curation team and outside experts to assemble and peer-review its pathway modules. The integration of ORCID within Reactome enables us to meet a key challenge with authoring, curating and reviewing biological information by incentivizing and crediting the external experts that contribute their expertise and time to the Reactome curation process. More information is available at ORCID and Reactome.

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Details on Person A single unattached kinetochore is capable of preventing cel...

Class:IdSummation:1598584
_displayNameA single unattached kinetochore is capable of preventing cel...
_timestamp2011-09-12 23:26:25
created[InstanceEdit:1597865] Higgins, JA, 2007-11-15
modified[InstanceEdit:1598195] Higgins, JA, 2009-03-05
[InstanceEdit:1601927] Preece, J, 2011-09-12
textA single unattached kinetochore is capable of preventing cells from exiting mitosis. The mitotic checkpoint provides a way for a localized defect to affect the global biochemical status of the cell. In principle, the signal that is generated at an unattached kinetochore diffuses throughout the cell to affect its target. There are currently two models for how this is achieved. One model is based on the observation that the Mad2 checkpoint protein binds and is rapidly released from unattached kinetochores. The kinetochore is believed to act as a catalyst that converts Mad2 into an inhibitory state that diffuses throughout the cell upon its release from the kinetochore. A second model proposes that the signal is amplified by a kinase cascade much like a conventional signal transduction pathway. This kinase cascade is believed to be comprised of the checkpoint kinases, hBUBR1, hBUB1, hMPS1. (imported from Human Reactome, Stable identifier: REACT_795.1)
(summation)[Pathway:1598627] Amplification of signal from the kinetochores [Arabidopsis thaliana]
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No pathways have been reviewed or authored by A single unattached kinetochore is capable of preventing cel... (1598584)