Query author contributions in Reactome
Reactome depends on collaboration between our curation team and outside experts to assemble and peer-review its pathway modules. The integration of ORCID within Reactome enables us to meet a key challenge with authoring, curating and reviewing biological information by incentivizing and crediting the external experts that contribute their expertise and time to the Reactome curation process. More information is available at ORCID and Reactome.
If you have an ORCID ID that is not listed on this page, please forward this information to us and we will update your Reactome pathway records.
Details on Person EGFR cancer mutants are assumed to recruit the PI3K complex ...
| Class:Id | Summation:1247350 |
|---|---|
| _displayName | EGFR cancer mutants are assumed to recruit the PI3K complex ... |
| _timestamp | 2018-11-28 19:29:30 |
| created | [InstanceEdit:1247348] Orlic-Milacic, Marija, 2011-04-01 |
| modified | [InstanceEdit:1247621] Orlic-Milacic, Marija, 2011-04-04 [InstanceEdit:1660492] Orlic-Milacic, Marija, 2011-10-13 [InstanceEdit:2008173] Orlic-Milacic, Marija, 2011-11-21 [InstanceEdit:2010566] Orlic-Milacic, Marija, 2011-11-22 [InstanceEdit:9630102] Orlic-Milacic, Marija, 2018-11-28 |
| text | EGFR cancer mutants are assumed to recruit the PI3K complex PIK3CA:PIK3R1 in a manner similar to wild-type EGFR. |
| (summation) | [Reaction:1226012] Binding of PI3K to complex of GRB2:GAB1 and ligand-responsive p-6Y-EGFR mutants [Homo sapiens] |
| [Change default viewing format] | |
No pathways have been reviewed or authored by EGFR cancer mutants are assumed to recruit the PI3K complex ... (1247350)
